Dioxins and furans are some of the most toxic chemicals known to science. A draft report released for public comment in September 1994 by the US Environmental Protection Agency clearly describes dioxin as a serious public health threat. The public health impact of dioxin may rival the impact that DDT had on public health in the 1960’s. According to the EPA report, not only does there appear to be no “safe” level of exposure to dioxin, but levels of dioxin and dioxin-like chemicals have been found in the general US population that are “at or near levels associated with adverse health effects.”
Agent Orange is linked to bladder cancer and hyperthyroidism:
Dioxin (a carcinogen) from Agent Orange is in GMO Plants and foods:
Dioxin causes Parkinsons disease:
The Ukranian president was poisoned by dioxin and nearly died:
Dioxin lasts for years inside of our bodies.
Once you are poisoned with this: it is extremely difficult to get rid of:
Dioxin lasts for decades in the environment. Long-term exposure to dioxins can cause harmful effects such as impairment of immune system, nervous system, endocrine system, and reproductive functions.
The HALF life of dioxin is 9-15 years and that is assuming no new contamination!
2,4-D is a pesticide that has been heavily used in agriculture all over the world for some fifty years or more. Alarmingly the ACP Evaluation has highlighted a large number of major data gaps-covering human health effects, aquatic and wider environmental risk. In addition to the number and the range of these data gaps, there continue to be concerns about long term adverse effects of 2,4-D on human health and water pollution.
Dow 2,4-Dichlorophenoxyacetic acid toxicity:
- Subchronic oral exposure to 2,4-D caused damage to the eye, thyroid, kidney, adrenals, and the ovaries and testes of laboratory animals.3,19 A subchronic NOEL was established at 15 mg/kg/day based on studies in rats.19 See the text box on NOAEL, NOEL, LOAEL, and LOEL.
- The chronic toxicity NOEL in rats and mice was determined to be 5 mg/kg/day in two-year studies.12,20 The maximum tolerated dose in the two-year rat study was 150 mg/kg/day in male rats and 75 mg/kg/day in females.20 Additional NOEL and NOAEL doses were 15 mg/kg for rats in a 90-day study, and 1 mg/kg for dogs in a 12-month study, respectively.12,21 Rabbits exhibited toxicity following dosing with either acid, salt, or ester forms of 2,4-D at doses of 30 mg/kg/day or greater.4 Chronic NOAELs and LOELs in dogs, however, varied for different parameters studied and by chemical form.21
- Rats showed no outward signs of toxicity following exposure to 200 mg/L of 2,4-D in drinking water for 30 and 100 days, but biochemical analysis suggested hepatic and muscle damage.17
- Researchers fed rats 2,4-D at doses of 1, 15, 100, and 300 mg/kg/day acid equivalents (ae). Changes in blood and thyroid parameters, organ weight ratios, and body weight gain were noted at 100 and 300 mg/kg/day doses.19 Chronic toxicity in the eye, kidney, thyroid and liver of the rat were similar to effects found in subchronic studies.20 Eye lesions were associated only with high doses of 150 mg/kg/day.20
- No human data were found on chronic effects of 2,4-D other than epidemiological studies of cancer occurrence. Although pesticide use has been linked to Parkinson’s disease and to respiratory disease in farmers, 2,4-D was not implicated in any relationships between pesticide exposure and subsequent disease.22,23 See the Carcinogenicity section below for more information on 2,4-D and cancer in humans. See the text box on Exposure.
Exposure: Effects of 2,4-D on human health and the environment depend on how much 2,4-D is present and the length and frequency of exposure. Effects also depend on the health of a person and/or certain environmental factors.
- Because 2,4-D has demonstrated toxic effects on the thyroid and gonads following exposure, there is concern over potential endocrine-disrupting effects.3 2,4-D is included in the U.S. EPA June 2007 Draft List of Chemicals for Tier 1 Screening.24
- No oncogenic effects were observed in rats or mice following 2 years of dietary exposure of 2,4-D with concentrations ranging from 5-150 mg/kg/day or 5-300 mg/kg/day, respectively.20 Similarly, researchers did not observe immunotoxic or oncogenic responses in dogs dosed with 1.0-7.5 mg/kg/day for either 13 weeks or 1 year.21
- A case-control study in companion dogs concluded that there was a “modest association” between malignant lymphoma in the dogs and the use of 2,4-D in their owners’ yards after accounting for other home and yard pesticide use.25 Other investigators have questioned the epidemiological association reported in that study.5,26
- Overall, there has been no consistent association between exposure to 2,4-D and tumor induction in animals.27 More recently, non-cytotoxic concentrations of 2,4-D were correlated to DNA damage and altered expression of some genes in hamster embryo cells.28
- The U.S. EPA evaluated 2,4-D for carcinogenic effects in 1988, 1992, and again in 2004. Each evaluation has concluded that “the data are not sufficient to conclude that there is a cause and effect relationship between exposure to 2,4-D and non- Hodgkin’s Lymphoma.” 2,4-D was categorized as “Group D – not classifiable as to human carcinogenicity” in 2004.3 See the text box on Cancer.
Cancer: Government agencies in the United States and abroad have developed programs to evaluate the potential for a chemical to cause cancer. Testing guidelines and classification systems vary. To learn more about the meaning of various cancer classification descriptors listed in this fact sheet, please visit the appropriate reference, or call NPIC.
- The International Agency for Research on Cancer (IARC), had not assigned 2,4-D a cancer rating as of June 2008. However, in 1987, IARC placed the family of chlorophenoxy herbicides in Group 2B, possibly carcinogenic to humans.29
- A discussion of the history of classification decisions regarding the carcinogenicity of 2,4-D has been published. A confounding factor in determining the carcinogenicity of 2,4-D is the frequent simultaneous exposure of workers to 2,4-D in addition to 2,4,5-T and its contaminant TCDD (dioxin), or to other herbicides. However, other work examining incidents of exposure to 2,4-D without simultaneous exposure to 2,4,5-T has found some association between 2,4-D and non-Hodgkin’s lymphoma.26
- Although the free acid form of 2,4-D did not damage chromosomes, there is limited evidence that commercial formulations may have the potential to do so.27 Overall, evidence for mutagenicity has been inconsistent.26,27,30
- Teratogenic effects were not observed in mice, rats, or rabbits unless the excretion capacity of the mother was overwhelmed following oral exposure to 2,4-D or its salt and ester forms.4,26 Reduced fetal viability was observed in hamsters following maternal dosing at 40 mg/kg/day during pregnancy, although effects did not follow a dose-response relationship.31
- Fetal abnormalities were observed in rats following oral doses of 90 mg/kg/day or greater beginning at fertilization; these doses were toxic to the mothers as well.4 A NOEL of 25 mg/kg/day was derived for fetal rats in one study, and a NOAEL of 12.5 mg/kg/day for the mothers and a developmental NOAEL of 50 mg/kg/day for the young were derived in another study.7 The overall maternal NOEL in rats was determined to be 8-17 mg/kg/day and overall developmental NOEL was 30 mg/kg/day 2,4-D acid equivalents.4
- Rabbit fetuses were unaffected at doses below 40 mg/kg/day administered to the dams although extra ribs were formed at doses above this threshold.4 In rabbits, the developmental NOEL was 30 mg/kg/day 2,4-D acid equivalents.4
- No experimental data are available regarding the effects of 2,4-D exposure on reproduction or development in humans. There are some reports of reproductive effects following occupational exposure to chlorophenoxy herbicides,7 including reduced sperm motility and viability following occupational exposure. Although motility and viability recovered over a period of several months, malformations were still present.32 Exposure to multiple pesticides in epidemiological studies make inference difficult.26
Hillary says to “eat up” it is “drought resistant”.
“Poison the world” Hillary will LIE to you to get you to eat this poison: